KAP1 stabilizes MYCN mRNA and promotes neuroblastoma tumorigenicity by protecting the RNA m6A reader YTHDC1 protein degradation

Abstract Background Neuroblastoma (NB) patients with amplified MYCN often face a grim prognosis and are resistant to existing therapies, yet MYCN protein is considered undruggable.KAP1 (also named TRIM28) plays a crucial role in multiple biological activities.This study aimed to investigate the relationship between KAP1 and MYCN in NB.Methods Trans

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